Teaching NeuroImages: Pseudo-optic disc edema from vitreopapillary traction

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vitreopapillary traction in non-arteritic anterior ischemic optic neuropathy

purpose: to describe the association between optic disc traction and non-arteritic anterior ischemic optic neuropathy (naion). methods: eighty three eyes of 83 patients with naion were evaluated by optical coherence tomography (oct) for detecting vitreous adhesion to the optic nerve head with separation from adjacent retina (partial posterior vitreous detachment). those who were negative for su...

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Vitreopapillary traction confirmed by optical coherence tomography.

1. Hardman JG, Limbird L, Gilman AG. The Pharmacological Basis of Therapeutics. 10th ed. New York, NY: McGraw-Hill; 2001. 2. Coulter DM, Clark DWJ, Savage RL. Celecoxib, rofecoxib, and acute temporary visual impairment. BMJ. 2003;327:1214-1215. 3. Lund BC, Neiman RF. Visual disturbance associated with celecoxib. Pharmacotherapy. 2001; 21:114-115. 4. Edwards IR, Biriell C. Harmonisation in pharm...

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Vitreopapillary traction in proliferative diabetic vitreoretinopathy

Aim—To present the clinical profile of a new entity in advanced proliferative diabetic vitreoretinopathy (PDVR). Mechanisms of vision loss due to vitreopapillary traction on the nasal optic disc are described, followed by an introduction of methods for prevention and treatment in such cases. Methods—17 patients with PDVR and traction on the nasal side of the optic disc, pallor of the optic nerv...

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Optic disc edema and optic nerve head drusen.

pletely. Several follow-up MRIs were performed. The most recent MRI, 1.5 years after the onset of symptoms, revealed an increase in number and extent of the white matter hyperintensities (Fig. 2). Glatiramer acetate therapy was initiated. On occasion, a one-and-a-half syndrome can be accompanied by a facial paresis if the fascicle or nucleus of the seventh cranial nerve in the lower part of the...

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Ten days after respiratory infection, a 32-year-old woman presented with headaches, ataxia, and diplopia without encephalopathy. Brain MRI revealed extensive white matter, brainstem, and cerebellar vasogenic edema, without gadolinium enhancement, partially regressive during follow-up (figure, A). CSF revealed transient elevated protein level (1.01 g/L) and hypercellularity (123 neutrophils/mm)....

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ژورنال

عنوان ژورنال: Neurology

سال: 2019

ISSN: 0028-3878,1526-632X

DOI: 10.1212/wnl.0000000000007785